FEEDING BROILERS FOR OPTIMUM GROWTH.

Chicks grow and develop at an incredible rate during the first week of hatching. With just 34-42 days from the beginning to end of the broiler production cycle, that makes the right choice of starter feed particularly critical.The importance of a feed that is specially tailored to the needs of chicks up to the age of ten days cannot be understated. The key consideration for the producer is the digestibility of the feed. Both the digestive tract and immune system are highly immature at hatching. So, unless the feed is easy to digest, most of the nutrients will pass through the digestive tract un absorbed and be excreted in the droppings. The chick will then draw energy from the immunoglobulins and unsaturated fatty acids in its yolk sac, leaving little for development and immunity.This is a serious problem for a chick that needs to develop vital organs and quadruple in weight by the time it is seven days old. The fact that chicks can go up to 48 hours from hatching to their first intake of feed is an added pressure.For all these reasons, a sub-optimal feed will only add to the challenges a chick has to overcome. Apart from impairing growth due to poor nutrient absorption, it will increase vulnerability to pathogenic bacteria and viruses, such as Salmonella and Campylobacter. A lot of nutrients are necessary to ensure proper growth. One of the most important of them is protein. Due to its high quality and lower cost than proteins from some animal sources, soy protein is a popular choice. Here, again, digestibility is the key to success. The issue with soy protein in its untreated form is its content of anti-nutritional factors (ANF). Though not a problem for older broilers, in the immature digestive system of a newly hatched chick ANF have a direct negative impact on protein absorption – and the chick's potential for healthy growth,the answer is reduction of the anti-nutritional factors to a very low level, while maintaining the nutritious protein components intact. An optimized starter feed that takes the immature digestive systems of hatchlings into account is another step in the ongoing optimization process, and also the reduction in rearing time. story credit; world poultry.

PSEUDO-RABIES!!!

A large number of swine-producing 
countries have eradicated the presence of pseudorabies, also known as Aujeszky’s Disease. Aujeszky’s Disease can be found throughout the world, especially in regions with dense swine populations including South America, Asia and Europe. Countries such as Germany, Austria, Sweden, Denmark, the United Kingdom, Canada, New Zealand and the United States have eradicated the disease from their domestic swine populations in the late nineties. China on the other hand has experienced an unprecedented outbreak since 2011. Vaccination proves to be an important tool to control PRV. Pseudo rabies virus (PRV), the causing agent of Aujeszky’s Disease, is an extremely contagious herpes virus that causes respiratory disease and reproductive problems, including abortions and stillbirths in breeding swine. In piglets, PRV can cause coughing, sneezing, fever, constipation, depression, seizures, ataxia, circling and excess salivation, with mortality in piglets less than one month of age being close to 100%. Occasional death losses in breeding and finishing pigs can also occur. The virus is transmitted through nasal and oral secretions, food, water, and the environment. It can also be carried on vehicle wheels, including tyres or buggy wheels, boots and clothing. The incubation period is commonly two to five days, with nasal and oral excretion and, in adult swine, vaginal, preputial, and/or milk secretion coincident or just preceding any primary symptoms. Additionally, adult Aujeszky positive swine may harbour the virus without showing clear signs. The virus can live in humid air and non-chlorinated water for up to seven hours; and in the soil, on clothing, and faeces for up to two days. Dogs, cats, and racoons can physically transmit the virus between farms, but usually the virus causes death in these animals. PR viruses comprise a single serogroup. However, both vaccine and wild-type viruses can be differentiated into groups by using combinations of physical and biological markers. Vaccination with modified live marker vaccines is a useful tool in controlling the disease and PRV has been eradicated in many countries by the use of modified live vaccines.

THE ROLE OF IRON IN PIGLET DEVELOPMENT.

Production methods have led to an impressive increase in 
productivity but have also increased iron needs in piglets. A potential iron gap can inhibit growth rates and 
profits significantly thus imposing threat can be eliminated simply by providing iron supplements/injections at early stages of development and over course of the piglet's life. Rapid development in modern swine production is a success , with piglets displaying incredible growth rates in their first weeks of life. However, the increased productivity has increased iron needs in piglets and, thereby, increased the risk of iron deficiency anaemia. Anaemia in piglets is a condition characterized by lack of haemoglobin in the body's red blood cells. Haemoglobin plays an important part in transporting oxygen from the lungs to the tissues of the body, and when the hemoglobin level is reduced, the body's access to the vital oxygen is reduced. The most common cause of anaemia in piglets is iron deficiency. Iron deficiency occurs when the piglet's own iron stores are depleted or when the exposure to stress conditions either inhibits the build-up of haemoglobin or increases the degradation of haemoglobin in the piglet. A critical time for iron deficiency is at weaning, when iron stores following the initial iron dextran injection in the first days of life may be depleted. The causes of low levels of iron in piglet;Increased litter sizes, result in lower iron stores at birth for the individual piglet and with the increased litter sizes, the already low levels of iron in the sow's milk are diminished to even lower levels. The fast growers are increasingly at risk of developing iron deficiency, as they face an increased risk of depletion of available iron stores due to their accelerated growth rates.The fast growers are often on an all-milk diet which offers a good source of all nutrients required by the piglet, except for iron. Weight gain on an all-milk diet is, therefore, associated with an increased risk of developing iron deficiency, which could eventually lead to iron deficiency anaemia. story credit; pig progress.

How to boost piglet immune response with vitamin D.

The benefits of vitamin D in pig feed go beyond the well-known function of calcium regulation and phosphorus homeostasis and its effect on bone development. Studies are showing that optimizing feed intake of vitamin D can boost piglet immunity in a number of different ways. The post-weaning phase is a critical period in a piglet's life. The development of a strong immune system at this early stage is key to securing its health and future optimum performance. However, a piglet has limited ability to mount and regulate an immune response when it is weaned from the sow at between three and five weeks old in commercial practice. Its immunity needs to develop as the passive protection from the sow's antibodies fade away and viral, bacterial and parasitic infections are at their highest risk. Any compromise to a piglet at this period has repeatedly been shown to impact negatively on its later performance. Vitamin D is an essential nutrient which is routinely added to animal feed, primarily because animals' blood levels of vitamin D vary considerably. It has become the focus of renewed attention by nutritionists and swine producers worldwide in recent years, because of findings that indicate that the benefits of vitamin D go beyond the function of the regulation of calcium and phosphorus homeostasis and its effect on bone development. Vitamin D metabolites control the expression of more than 200 genes through activation of the vitamin D receptor, which regulates or modulates gene expression within the target cell. This gives the vitamin a role in many functions in swine, including immunity, muscle function and reproduction. The vitamin D receptor is not only found in the intestinal enterocyte, the osteoblast, and the renal cells, but it is also found in a wide range of cell types whose function does not relate to calcium metabolism, such as the intestine, pancreas, heart, eye, brain, thyroid, parathyroid, muscle, or immune cells. The newborn piglet is exposed to a vast array of antigens from the moment it is born. It relies on maternally-derived immunity for protection, until it develops its own active immunity. Prior to weaning, sow's milk provides most of the nutrients that the piglet will receive for maximal growth and health. However, sow's milk provides little vitamin D. Supplementing sows with vitamin D before birth could provide a nutritional strategy to increase vitamin D status of the piglet, through placental transport or via sow's milk. story credit; pig progress.

"African Swine Fever is a man-made disease"

Dr Klaus Depner and Dr Sandra Blome, of the Friedrich-Loeffler-Institut in Germany have new insights about the virus , they have studied cases and are of the opinion that “Most problems are a matter of human misbehavior.”

 African Swine Fever, it should be that the major threat with regard to the virus is not the virus itself, but how humans deal with it.Trials at the FLI showed that there is no difference in the way the ASF virus affects wild boars or domestic pigs.

Logically, one of the major questions that the researchers had when ASF was introduced in 2007 in the Caucasus was: how would the virus spread and behave in wild boars? Depner: “Essentially, we had two hypotheses.

The first one was that the disease in wild boars would die out due to the high virulence of the virus.”Blome: “Roughly, the animals get sick four days after infection.”Depner: “Usually death will follow within three to six days, .

This means that almost all infected hosts will die very quickly, which means that the virus will cease to exist very soon because it kills its host.

In that case, we would not have to worry, ASF would do its job extinguishing itself.”Blome, however, points to the fact that ASF virus is not that contagious. High viral loads are found in blood, but saliva or faeces contain less virus:

“We overestimated the contagiousness of African Swine Fever. The disease moves very slowly. When looking at affected wild boar populations, most of them have not been significantly reduced. The virus doesn’t spread that quickly at all.” Blome adds, “Wild boars shed the virus mainly when they are very sick and in the final stage of the disease.

When the animals have high fever it’s in their character to stay where they are, and they are certainly not going to walk very far when they feel bad.”Depner: “So what we have here is a virus that is very stable in its environment without fast movement. It neither dies out, nor moves.

 Undisposed carcasses of infected wild boars remain infectious for a long time in the environment and become a source of infection for healthy animals.” The human factor; its usually a case of human misbehavior. What happened is that infected meat made it to the market.

When many pigs started to die, they were sent to slaughter. Pig prices dropped, cheap meat entered the market and the meat made its way into homes – and into suitcases. This is how the virus dispersed.

 The virus spread along the main roads, the transport routes. This spread bears a 100% human mark.” Humans can be identified as having aggravated the situation ever since as well.

 Since wild boars have often been thought to be spreading the virus, in several countries attempts were launched to eradicate them – Poor bio security protocols have also been identified as cause of spread of the virus.

Bio security measures include the following; Changing clothes, working hygienically and making sure nothing from the outside reaches the inside.

 Story credit; world poultry.

POULTRY PRODUCTION AND CLIMATE CHANGE.

A team of researchers from US University of Delaware traveled to Africa in 2012 to search for exemplary chickens that could survive a hotter planet. The purpose was to develop new breeds of farm animals that can stand up to the hazards of global warming. Heat-resistant breeds of farm animals will be essential to feeding the world as climate change takes hold. This means that efforts such as trying to map the genetic code of African naked-neck chickens to find out if their ability to withstand heat can be bred into broiler flocks.Warmer temperatures can cause a lot of problems for animals like turkeys as these are vulnerable to a condition that makes their breast meat mushy and unappetizing. Turkeys are not heat tolerant at all, and when heat waves strike their breast muscles problems. According to Gale Strasburg, a professor of food science and human nutrition at Michigan State University Within a day or two after the heat wave hits, you will go from there being no problem at all on a farm to 40% of turkey breasts having a problem. Heat waves and temperature changes linked to breed susceptibility are points of focus for research on producing more heat resistant breeds to ensure food security.Work is ongoing on breeding techniques to incorporate the heat tolerant strains into lines of production.

ADDITIVES ; BETTER ALTERNATIVES TO ANTIMICROBIAL GROWTH PROMOTERS IN POULTRY.

The demand for healthier safety-proven production methods and the trend to reduce antibiotic uses is undeniable, and is steadily expanding. Simultaneously, the increasing ineffectiveness of antibiotics due to resistance 
development is another concern. The concern about antibiotic residues in meat products is solved in many countries, but the bacterial resistance to antibiotics is a much more complex problem, involving the entire medical profession. In animal production—poultry production in particular :ceasing the use of antibiotics as growth promoters has changed the technical approaches used to counter the removal or reduction of these products. Without antibiotics, the microbiota in the birds’ gastrointestinal tract must be viewed as an entity to implant, develop and control to ensure that the animals are healthy and can grow according to their genetic potential. The removal of antibiotic growth promoters has revealed that it is crucial to manage the makeup of the birds' intestinal microbiota to avoid or at least limit the risks of health problems inherent to intensive production, hence alternatives are necessary to ensure health of birds and food safety. The main strategies can be outlined as follows: Selectively introduce favourable bacterial populations from a very young age; Provide a regular supply of nutrients specific to the beneficial bacteria; Act directly on pathogenic bacteria. Different methods are available, such as the dietary supplementation with probiotics, prebiotics, organic acids and essential oils. These products act either avoiding bacterial adhesion to the intestinal cells or through bactericidal or bacteriostatic effects. The goal is to achieve the most stable balance on the microbiota in order to avoid the trouble caused by bacteria like E. coli, Clostridium and Salmonella. The gastrointestinal microbiota profile depends on numerous factors such as the health of the breeding stock, production system, stressors (vaccination, viral episodes, etc.), nutrition and stocking densities. The first bacteria to colonise the animals’ digestive tract will shape the intestinal ecosystem for the introduction of global intestinal microbiota. Selectively colonising the gastointestinal tract with beneficial bacteria can modulate the expression of certain genes in the tract’s epithelium, creating conditions for establishing beneficial microbiota. Since the first micro-organisms that come into contact with a newly hatched chick’s gastrointestinal tract are from the breeding stock, controlling the microbiota in the parents would be ideal. Studies have demonstrated that the use of beneficial bacteria in low doses in the hatchery improves chicks’ resistance to pathogens. Other studies have validated that an in-ovo injection of FOS (fructooligosaccharide) helps maintaining higher levels of bifidobacters, with positive effects on zootechnical performance and mortality rate. Most of the microbial strains in probiotics are of the genus Bifidobacterium or Lactobacillus. In certain conditions, lactobacilli can produce metabolites that limit the growth of Salmonella by modulating immunity and avoiding bacterial binding to the epithelial cells of the intestine. However, according to some publications, this type of effect is limited. In poultry production, interactions between the birds’ feed and their intestinal health have been amply demonstrated. In the past, the use of antibiotic growth enhancers had the potential to mask a number of problems. In the field, functional diet-based strategies need to be adapted to the different sanitary and production contexts for each production system. Selectively introducing favourable bacterial populations for young chicks, providing a regular supply of nutritious substrates specific to the beneficial bacteria and effectively controlling pathogenic bacteria are important courses of action to ensure the intestinal health of the birds via their feed. Prebiotics, enzymes, and combinations of organic acids and essential oils can be viable alternatives to antimicrobials. story credit; world poultry.