VETERINARY MEDICINE: Dilated Cardiomyopathy in Dogs (DCM).

VETERINARY MEDICINE: Dilated Cardiomyopathy in Dogs (DCM). Dilated Cardiomyopathy (DCM) is one of the most common acquired heart diseases in dogs. This disease is rarely diagnosed in cats or small-breed dogs; however, it is a common cause of heart disease in large and giant breed dogs, and usually occurs more in those that are middle- to older-aged. DCM is a condition where the heart muscle (myocardium) loses its ability contract normally and as effectively . Although DCM can affect both sides of the heart simultaneously or separately, myocardial failure of the left side is most common. Since the heart muscle cannot work as efficiently to pump blood out of the heart, blood backs up within the heart chambers and the heart enlarges in size. If pressures on the left side of the heart become significantly high as a result of increased blood volume, left-sided congestive heart failure or pulmonary edema (fluid within the lungs) can result. Although less common, myocardial failure of the right side of the heart can also occur. Similar volume overload of the right heart may result in right-sided congestive heart failure, often resulting in excessive free-fluid in the abdomen (ascites) and/or chest (pleural effusion). A familial or genetic component is believed to exist in the majority of cases. This is because of the prevalence of the disease in specific breeds such as the Doberman Pinscher, Great Dane and Boxer. The signs of DCM vary depending on the breed of dog and stage of the disease. Loss of appetite, pale gums, increased heart rate, coughing, difficulty breathing, periods of weakness, and fainting are signs commonly seen. Since blood (plasma) is being backed up into the lungs, respiratory signs are usually due to pulmonary edema and/or heart enlargement. Blood returning to the right side of the heart from the body may also back up leading to fluid accumulation in the abdomen (ascites) or in the chest cavity (pleural effusion). Weakness or collapse may be caused by abnormal heart rhythms (arrhythmias) and poor distribution of blood (depressed cardiac output). Treatment is tailored based on clinical presentation of each individual patient.

Moments of intense joy can damage your heart.

Even in heartbreak, pain, misery and ennui, brief episodes of happiness can buoy us, inspire us and give our lives meaning. They can also, according to a new study, cause the left ventricle of the heart to suddenly weaken. Unfortunately for fans of joy, a report from the University of Zurich has found that moments of intense happiness can cause breathlessness and chest pains. This is because they can induce takotsubo cardiomyopathy, a heart condition that causes the left ventricle to temporarily weaken. It's normally caused by stress, anger, grief or fear -- around 75 percent of reported cases come from strong negative feelings. According to the study, around one in twenty cases are caused by joy. Problematic incidents included birthday parties, weddings, a reunion between two long-estranged friends, winning a jackpot and an all-clear from a CT scan and 95 percent of the patients were post menopausal women. Findings from the study were published in the European Heart Journal. "We've shown that the triggers for takotsubo syndrome can be more varied than previously thought," said Dr Jelena Ghadri, who worked on the report. "A takotsubo syndrome patient is no longer the classic 'broken-hearted' patient, and the disease can be preceded by positive emotions too." "Clinicians should be aware of this and also consider that patients who arrive in the emergency department with signs of heart attacks, such as chest pain and breathlessness, but after a happy event or emotion, could be suffering from takotsubo syndrome just as much as a similar patient presenting after a negative emotional event," Ghadri added. Luckily for victims of the syndrome, it's only temporary -- after treatment is given, the heart returns to normal after a few weeks. culled from wired.co.uk

POLLUTION AND THE RISK OF STROKE.

A report by the American Heart Association, states that stroke may be associated with climate change and air pollution. In a study conducted in the United States and China, the total number of strokes increased as pollution levels rose. Changes in climate may have contributed to the level of air pollution in a region, which in turn contributed to the total number of strokes. The higher pollution levels were linked to a higher total number of strokes, and researchers said it reaffirmed the growing evidence that climate change and overall air quality contributes to cardiovascular disease, according to research presented at the American Stroke Association's International Stroke Conference 2016. The study, which used data from the United States and China, is one of the first to investigate the interaction between air quality and the number of stroke cases (prevalence) along with the potential effect of temperatures on the association. Across the two countries, researchers found that the total number of stroke cases rose 1.19 percent for each 10 micrograms per cubic meter of air (µg/m3) increase of PM2.5. In addition, Liu said researchers found a significant regional variation in PM2.5 levels that was linked to the number of stroke cases.Researchers also found that temperature had an impact on air quality and risk of stroke. Patients with stroke are in danger of dehydration due to high temperatures in the summer, and are in danger of suffering from pneumonia, influenza and other respiratory diseases in winter. Women and the elderly also appear more vulnerable to stroke risk due to air quality and heat-related diseases. Stroke is the fifth leading cause of death in the United States killing nearly 129,000 people every year, and is a leading cause of disability. Worldwide, the prevalence of stroke stood at 33 million, with 16.9 million people suffering their first stroke, and is the second-leading cause of global death behind heart disease. Although patients cannot control air quality, Liu said, the findings provide evidence for policy makers and public health leaders to develop better models for monitoring and predicting climate changes so that patients can better protect themselves.

CALF BORN WITH HEART IN THE NECK!!!.

The year has been filled with spectacular breakthroughs in science especially in animal health and production,also strange discoveries were uncovered.One of the strange occurrence is the calf born with heart in the neck. This rare case was reported by the Observer-reporter Washington Co.;Tom Leech had never seen anything like it in his 15 years living at Longview Farms in Amwell Township. There was something unusual about the newborn Shorthorn bull he brought in from the frigid air one Saturday in March.He warmed up the calf with a blow dryer and electric blanket, which warded off hypothermia. But when he touched his neck, he felt a pulsation. Ba-boom, ba-boom, ba-boom.It was his heart.!!!.“No one has ever seen it, never heard of it,” said Leech, who owns the family farm with his wife, Debbie. The calf’s mother, G.I. Jane, has birthed healthy calves in the past, and so has the father. None of the calves born at Longview, a cattle-breeding farm, has had any genetic disorders or abnormalities.But the 6-week-old youngster, dubbed Cardio Brisket, is a “unique calf,” Debbie said. Dr. Todd Moores of Wheeling Veterinary Associates has seen two-headed calves before, but never one with a heart in its neck.“I could tell by looking at it. You can see the heart beating right there,” Moores said. “It even makes a noise because there’s fluid around it, so it makes a sloshing noise.”He said Brisket appears to have a defect in his thoracic inlet, and he suspects that his sternum never developed properly. Several ribs may be missing, as well. The opening allowed the heart to prolapse into the neck region, and while it can be pushed down, it just “flops out” again, Tom said. Researchers at the Ohio State College of Veterinary Medicine offered to take Brisket for testing, but they couldn’t guarantee his survival, so the Leeches declined. Besides, they were starting to consider Brisket a valuable member of their farm family. Brisket stays in the barn, apart from the other 20-some cattle. He is taken out separately during the day to get exercise.“They’re like kids,” Tom said. “They start playing, they start butting, and I’m afraid they’ll butt his chest and could possibly injure his heart.”Moores said it’s hard to tell how long Brisket could live. He has a heart murmur, which could lead to complications when he grows bigger. The Leeches said they will keep him as long as they can, though. They don’t want to take him to the fair and display him like a “circus attraction,” Debbie said, but they wanted to document the case for future reference. Despite the complications, Brisket appears to be getting by. He just started eating grain and has no problem swallowing the food.“He’s healthy, but all the people I’ve talked to said, ‘You’ve got to put him down,’” Tom said. “If he was suffering, we would probably do something with him, but he’s not.” There has been 2 other documented cases of such abnormality ;one in Kentucky in 1903, and another in Turkey, but the date was unknown.